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| | #31 | ||
| Senior Member+ | Quote:
__________________ ♥ Jen I know the answer! The answer lies within the heart of all mankind! The answer is twelve? I think I'm in the wrong building. - Charles M. Schulz Quote:
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| | #33 |
| Full Member |
humm....well I researched the heck out of it when my guy had it and everything said "only white feet will get this " maybe you had something else... not scratches ???
__________________ Mommy of: 12 yr old QH Buckskin: Cash |
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| | #34 | |
| Senior Member+ |
No, I have most definitely had (start to finish) scratches on dark legs. As many as light, easily. It's a bacterial infection... and while less pigmented skin may be slightly more susceptible, it happens quite often on dark legs.
__________________ ♥ Jen I know the answer! The answer lies within the heart of all mankind! The answer is twelve? I think I'm in the wrong building. - Charles M. Schulz Quote:
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| | #35 |
| Senior Member | Nope. Had a vet look at my mare when she had scratches... she only had it on her dark legs. And yes it WAS scratches. Old wives tale that it only occurs on white legs.
__________________ Sexy by Christmas Challenge-r!! lbs Lost: 0 |
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| | #36 |
| Full Member |
Or maybe it was that white legs/feet are more "prone" to it ??? Now that I think about it I believe that is what I read. It usually starts with dry skin or a cut/abrasion of some sort... and then it gets infected with the mud... funny how my guy's hind feet are white and only got the scratches...the dark front feet were standing in the same mud... almost like photosynthesis... sensitive skin back there....
__________________ Mommy of: 12 yr old QH Buckskin: Cash |
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| | #38 | |
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| | #39 |
| Full Member Join Date: Oct 2009
Posts: 35
![]() | Photosensitazation / alsike clover poisoning
This tends to show up on the white areas of the lower legs and face. Secondary photosensitization in a horse on pasture: presumptive alsike clover poisoning Sarah Hann November 4th, 2009 Preclinical Advisor - Dr. Marnie Fitzmaurice Clinical Advisor - Dr. Mary Smith A 2-year old, gelded, Chincoteague Pony was presented to the Ambulatory Service on Friday, 5/29/09 for lethargy, anorexia, and mild colic of one-day duration. The colic was characterized by repeated episodes of recumbency without rolling. On presentation, the gelding was recumbent and lethargic with a flaccid lower lip, a deviated muzzle, and decreased tongue tone. The pony had a decreased menace response with a normal pupillary light reflex but showed no evidence of ataxia or decreased proprioception. The gelding was afebrile and eupneic, with normal gut sounds in all four quadrants. Primary differentials at the time included trauma or colic with subsequent facial nerve paralysis; however, causes of brainstem and cranial nerve signs could not be ruled out due to the presence of decreased tongue tone. The gelding was given a dose of flunixin meglumine and the owners were instructed to monitor clinical signs. The following day, Saturday 5/30/09, the Ambulatory Service was called to evaluate continued signs of mild colic. On presentation the second day, ptosis was present OS. The muzzle deviation had resolved but the white snip was swollen, edematous, and blistering. In addition, the gelding had icteric mucous membranes. The pasture was examined and found to contain alsike clover, Trifolium hybridum. Blood was drawn that day and evaluated on Monday, 6/1/09. Significant biochemical abnormalities included increased aspartate aminotransferase, sorbitol dehydrogenase, glutamate dehydrogenase, alkaline phosphatase, and gamma-glutamyl transferase as well as increased direct and indirect bilirubin. Treatment was instituted to support liver function and palliate secondary photosensitization. The gelding was removed from the pasture containing alsike clover and housed indoors during the day to avoid ultraviolet light exposure. The pony received anti-endotoxic doses of flunixin meglumine and a course of trimethoprim sulphamethoxazole; blisters were treated with zinc ointment and triple antibiotic. Hay was provided ad libitum and supplemented with a high fat, low carbohydrate grain with the addition of 2000 IU of vitamin E daily. Problem List: Secondary photosensitization Hepatic encephalopathy Hepatic dysfunction Selected References: Colon JL, Jackson CA, Del Piero F. Hepatic dysfunction and photodermatitis secondary to alsike clover poisoning. Comp Cont Educ Pract Vet 1996; 18(9): 1022-1026. Nation, PN. Hepatic disease in Alberta horses: A retrospective study of "Alsike clover poisoning" (1973-1988). Can Vet J 1991; 32: 602-607. Talcott, P. Alsike Clover (Trifolium hybridum) and Red Clover (Trifolium pratense) Poisoning. in Robinson NE, Sprayberry KA: Current Therapies in Equine Medicine. Philadelphia: Saunders, 2008; pp 214-224, 790-791. |
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| | #40 |
| Senior Member+ |
Oops, meant to post an update.. No pictures! But I followed some of the directions and it's cleared up like crazy! We will see how it does as its been raining off and on today and I threw him outside... But I really do think it's related to the moisutre though.. I didn't clip his legs, no way to but oh well they're not that curly yet anyways. |
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